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This interaction leads to the dephosphorylation of IκB kinase, preventing the activation of NF-κB, a transcription factor that regulates genes involved in immune responses, inflammation, and cell survival ( Through these combined mechanisms, ICP34.5 serves as a critical factor in HSV-1 pathogenesis by supporting viral replication, evading multiple immune pathways, and altering host cellular functions

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The results obtained in the course of this study showed significantly elevated values of TOS and OSI, increased levels of lymphocyte DNA damage, and an elevated MDA concentration in migraine patients as compared with the control