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Third, many treatment pairs include two studies or at the most a maximum of three studies, which were not sufficient to correctly assess the transitivity assumption

L-Glutamine

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Neuron-specific deletion of Gls1 in mice results in impaired glutamatergic neurotransmission, respiratory dysfunction, and early postnatal lethality, underscoring its non-redundant physiological role in the central nervous system [53,54,55]