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Given the central role of the JAK/STAT pathway in the pathogenesis of AA, it represents a therapeutic target that has opened new avenues for understanding and managing the disease.5 Currently, two therapies have been approved for severe AA: baricitinib, an oral JAK1/2 inhibitor, approved for the treatment of adults (≥18 years),9 and ritlecitinib, an oral dual selective inhibitor of the JAK3/TEC kinase family, approved for patients aged ≥12 years

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Common features between targets across diseases permit the repositioning of already developed drugs effective for one disease to be assessed in another, as epitomized by exendin-4 – an efficacious drug in type 2 diabetes mellitus [184-186, 188] presently being assessed in early efficacy studies in PD and AD